Disease

Alzheimer's disease is a neurodegenerative disease that usually starts slowly and progressively worsens, & is the cause of 60–70% of cases of dementia. The most common early symptom is difficulty in remembering recent events. As the disease advances, symptoms can include problems with language, disorientation (including easily getting lost), mood swings, loss of motivation, self-neglect, & behavioral issues. As a person's condition declines, they often withdraw from family & society. Gradually, bodily functions are lost, ultimately leading to death. Although the speed of progression can vary, the average life expectancy following diagnosis is three to twelve years. Amyloid plaques build up in Alzheimer's brains . Obesity increases 3x the risk of Alzheimer's Plaques form when protein pieces called beta-amyloid clump together. Beta-amyloid comes from a larger protein found in the fatty membrane surrounding nerve cells. Beta-amyloid is chemically "sticky" and gradually builds up into plaques. Beta amyloids comes from LDH cholesterol Amyloid beta (Aβ or Abeta) denotes peptides of 36–43 amino acids that are the main component of the amyloid plaques found in the brains of people with Alzheimer's disease. The peptides derive from the amyloid-beta precursor protein (APP), which is cleaved by beta secretase and gamma secretase to yield Aβ in a cholesterol-dependent process and substrate presentation. Beta-amyloid comes from a larger protein found in the fatty membrane surrounding nerve cells. Beta-amyloid is chemically "sticky" and gradually builds up into plaques. The brain shrinks with age due to a lack of HDL high density lipo protein omega 3 Although a unified theory for microbial colonization of the brain has not been formed, multiple research groups have suggested ways this occurs. Weber et al noted that specific species of bacteria identified in studies exploring the brain microbiome in AD are normally found in the oral microbiome. Therefore, they hypothesized, that pathogenic changes in the oral cavity (often seen in AD) may damage connective tissues. This tissue destruction releases bacteria from the oral cavity, allowing for nervous system infection. Some of these bacteria can create a biofilm, through the production of amyloid proteins. These bacterial amyloids share similarities to the disease-causing versions.13 These amyloids may then allow other native amyloid proteins to aggregate and form colonies, beginning the pathogenesis of AD.
https://www.youtube.com/watch?v=zTd0-A5yDZI
Inside Alzheimer’s disease
https://www.youtube.com/watch?v=YMwZIqWQl-k
Alzheimer's Disease: Latest Research and Prevention Strategies
https://www.youtube.com/watch?v=IG_iwNY3woI
Transmission of misfolded proteins in neurodegenerative disorders (Dr. Virginia Lee}
https://www.youtube.com/watch?v=BW2zN3J2lfs
ALZHEIMER'S - CAN WE PREVENT IT?
https://www.youtube.com/watch?v=k_P7Y0-wgos
The Man With The Seven Second Memory

Antibiotics are dangerous & bad for the microbiome antibiotics kill off good bacteria consequently making the organism host fat & obese that's why animals which are given antibiotics become fat especially pigs . it turns out antibiotics make pigs less healthy phage therapy should be adopted
https://www.youtube.com/watch?v=OfCNfmvWEio
Why Are Antibiotics And Vaccines Failing Us? [4K] | The Battle Against Superbugs | Spark
https://www.youtube.com/watch?v=rERHPiYxc64
The Largest Civilization in The World
https://www.youtube.com/watch?v=Lkh84w497sY
Battle Against Bacteria: A Race Against The 'Superbug' | Disease Hunters | Part 2/3
https://www.youtube.com/watch?v=EkyAuG9RSSU
When Antibiotics Don't Work (full documentary) | FRONTLINE
https://www.youtube.com/watch?v=NlOGh5lSYtY
Antibiotic-resistant bacteria projected to kill 39 million by 2050, report says • FRANCE 24

Avian flu Avian influenza, also known as avian flu or bird flu, is a disease caused by the influenza A virus, which primarily affects birds but can sometimes affect mammals including humans. Wild aquatic birds are the primary host of the influenza A virus, which is enzootic (continually present) in many bird populations. Symptoms of avian influenza vary according to both the strain of virus underlying the infection, and on the species of bird or mammal affected. Classification of a virus strain as either low pathogenic avian influenza (LPAI) or high pathogenic avian influenza (HPAI) is based on the severity of symptoms in domestic chickens and does not predict severity of symptoms in other species. Chickens infected with LPAI display mild symptoms or are asymptomatic, whereas HPAI causes serious breathing difficulties, significant drop in egg production, and sudden death. Domestic poultry may potentially be protected from specific strains of the virus by vaccination. Humans and other mammals can only become infected with avian influenza after prolonged close contact with infected birds. In mammals including humans, infection with avian influenza (whether LPAI or HPAI) is rare. Symptoms of infection vary from mild to severe, including fever, diarrhoea, and cough. Influenza A virus is shed in the saliva, mucus, and feces of infected birds; other infected animals may shed bird flu viruses in respiratory secretions and other body fluids (e.g., cow milk). The virus can spread rapidly through poultry flocks and among wild birds. A particularly virulent strain, influenza A virus subtype H5N1 (A/H5N1) has the potential to decimate domesticated poultry stocks and an estimated half a billion farmed birds have been slaughtered in efforts to contain the virus.
https://www.youtube.com/watch?v=FYSnR8w9yV0
Silent Spreaders: Unmasking the Deadly Avian Influenza Virus | Documentary

Beta Amyloid plaques (also known as neuritic plaques, amyloid beta plaques or senile plaques) are extracellular deposits of the amyloid beta (Aβ) protein mainly in the grey matter of the brain. Degenerative neuronal elements and an abundance of microglia and astrocytes can be associated with amyloid plaques. Some plaques occur in the brain as a result of aging, but large numbers of plaques and neurofibrillary tangles are characteristic features of Alzheimer's disease. The plaques are highly variable in shape and size; in tissue sections immunostained for Aβ, they comprise a log-normal size distribution curve, with an average plaque area of 400-450 square micrometers (μm2). The smallest plaques (less than 200 μm2), which often consist of diffuse deposits of Aβ, are particularly numerous. Plaques form when Aβ misfolds and aggregates into oligomers and longer polymers, the latter of which are characteristic of amyloid.

Candida is a horrible fungus there are several non prescription supplements to get rid of Candida the best copper & Hydrogen Peroxide do a really good job killing off candida bacteria; drink caprylic acid, hydrogen peroxide, Fluorine ,Chlorine & copper to kill candida the strongest candida killers are generally recognized to be antifungal drugs, such as fluconazole (brand name Diflucan C13H12F2N6O ), nystatin C47H75NO17, and clotrimazole C22H17ClN2 .
https://www.youtube.com/watch?v=HMgqnzWXF6k&t=529s
Candida: The Invisible Disease of 21st Century (A Documentary)

Chemotherapy is dangerous most people don't survive past the side effects of chemotherapy i know many people who died because of the side effects of chemotherapy youre better off fighting cancer with immunotherapy and supplementation 
https://www.youtube.com/watch?v=RgWQCGX3MOk
How does chemotherapy work? - Hyunsoo Joshua No

KRAS mutations are present in about 40–45% of Colon Cancer cases. Vitamin c targets KRAS to cure colon cancer take supplements as Hydrogen Peroxide Vitamin C & Nitric Oxide & have a Hydrogen waterbottle 

Coronavirus disease 2019 (COVID-19) is a contagious disease caused by the coronavirus SARS-CoV-2. The first known case was identified in Wuhan, China, in December 2019. Most scientists believe the SARS-CoV-2 virus entered into human populations through natural zoonosis, similar to the SARS-CoV-1 and MERS-CoV outbreaks, and consistent with other pandemics in human history. Social and environmental factors including climate change, natural ecosystem destruction and wildlife trade increased the likelihood of such zoonotic spillover. The disease quickly spread worldwide, resulting in the COVID-19 pandemic. The symptoms of COVID‑19 are variable but often include fever, fatigue, cough, breathing difficulties, loss of smell, and loss of taste. Symptoms may begin one to fourteen days after exposure to the virus. At least a third of people who are infected do not develop noticeable symptoms. Of those who develop symptoms noticeable enough to be classified as patients, most (81%) develop mild to moderate symptoms (up to mild pneumonia), while 14% develop severe symptoms (dyspnea, hypoxia, or more than 50% lung involvement on imaging), and 5% develop critical symptoms (respiratory failure, shock, or multiorgan dysfunction). Older people are at a higher risk of developing severe symptoms. Some complications result in death. Some people continue to experience a range of effects (long COVID) for months or years after infection, and damage to organs has been observed. Multi-year studies are underway to further investigate the long-term effects of the disease. COVID‑19 transmission occurs when infectious particles are breathed in or come into contact with the eyes, nose, or mouth. The risk is highest when people are in close proximity, but small airborne particles containing the virus can remain suspended in the air and travel over longer distances, particularly indoors. Transmission can also occur when people touch their eyes, nose or mouth after touching surfaces or objects that have been contaminated by the virus. People remain contagious for up to 20 days and can spread the virus even if they do not develop symptoms. Testing methods for COVID-19 to detect the virus's nucleic acid include real-time reverse transcription polymerase chain reaction (RT‑PCR), transcription-mediated amplification, and reverse transcription loop-mediated isothermal amplification (RT‑LAMP) from a nasopharyngeal swab. Several COVID-19 vaccines have been approved and distributed in various countries, many of which have initiated mass vaccination campaigns. Other preventive measures include physical or social distancing, quarantining, ventilation of indoor spaces, use of face masks or coverings in public, covering coughs and sneezes, hand washing, and keeping unwashed hands away from the face. While drugs have been developed to inhibit the virus, the primary treatment is still symptomatic, managing the disease through supportive care, isolation, and experimental measures.
https://www.youtube.com/watch?v=5DGwOJXSxqg
COVID-19 Animation: What Happens If You Get Coronavirus?

Diabetes mellitus, often known simply as diabetes, is a group of common endocrine diseases characterized by sustained high blood sugar levels. Diabetes is due to either type 1 diabedets; the pancreas not producing enough insulin, or type 2 diabetes; the cells of the body becoming unresponsive to the hormone's effects. Classic symptoms include thirst, polyuria, weight loss, and blurred vision. If left untreated, the disease can lead to various health complications, including disorders of the cardiovascular system, eye, kidney, and nerves. Untreated or poorly treated diabetes accounts for approximately 1.5 million deaths every year. The major types of diabetes are type 1 and type 2. The most common treatment for type 1, insulin replacement therapy (insulin injections), while anti-diabetic medications (such as metformin and semaglutide) and lifestyle supplement can be used to manage type 2. Gestational diabetes, a form that arises during pregnancy in some women, normally resolves shortly after delivery. As of 2021, an estimated 537 million people had diabetes worldwide accounting for 10.5% of the adult population, with type 2 making up about 90% of all cases. It is estimated that by 2045, approximately 783 million adults, or 1 in 8, will be living with diabetes, representing a 46% increase from the current figures. The prevalence of the disease continues to increase, most dramatically in low- and middle-income nations. Rates are similar in women and men, with diabetes being the seventh leading cause of death globally. The global expenditure on diabetes-related healthcare is an estimated US$760 billion a year. Taurine & hydrogen peroxide are supplements which cue diabetes you simply have to take a lot of taurine to cure diabetes obesity leads to diabetes & cancer saturated fats activate TLR4 which synthesise ceramides and ceramides block the insulin cascade causing insulin resistance Up to 70% of people with type 2 diabetes also have nonalcoholic fatty liver disease (NAFLD), the most common chronic liver disease in people with type 2 diabetes. Metformin C4H11N5 & Rapamycin C51H79NO13 are two leading diabetes drugs 
https://www.youtube.com/watch?v=GYM9WsqNtiQ
Can Genetically Modified Lettuce Cure Diabetes?
https://www.youtube.com/watch?v=DMoh8_qGK30
How diabetes destroys the human body
https://www.youtube.com/watch?v=HJGjNTJgf48
How insulin works
https://www.youtube.com/watch?v=14sTJNrUA2c
What Diabetes Does to the Body | Can You Reverse It?
https://www.youtube.com/watch?v=x6EOhTzVKsE
Diabetes & Mitochondria: How Nutrition Connects the Two. [Study 32]
https://www.youtube.com/watch?v=RqZwxQLKsDc
Do This To ACTUALLY Reverse Diabetes Naturally In 4 Weeks! Step by step.
https://www.youtube.com/watch?v=kzC-OYlz6d8
The Fastest Way To Reverse Fatty Liver - Results In Days!

Glioblastoma, previously known as glioblastoma multiforme (GBM), is the most aggressive and most common type of cancer that originates in the brain, and has a very poor prognosis for survival. Initial signs and symptoms of glioblastoma are nonspecific. They may include headaches, personality changes, nausea, and symptoms similar to those of a stroke. Symptoms often worsen rapidly and may progress to unconsciousness. The cause of most cases of glioblastoma is not known. Uncommon risk factors include genetic disorders, such as neurofibromatosis and Li–Fraumeni syndrome, and previous radiation therapy. Glioblastomas represent 15% of all brain tumors. They are thought to arise from astrocytes. The diagnosis typically is made by a combination of a CT scan, MRI scan, and tissue biopsy. There is no known method of preventing the cancer. Treatment usually involves surgery, after which chemotherapy and radiation therapy are used. The medication temozolomide is frequently used as part of chemotherapy. High-dose steroids may be used to help reduce swelling and decrease symptoms. Surgical removal (decompression) of the tumor is linked to increased survival, but only by some months. Despite maximum treatment, the cancer almost always recurs. The typical duration of survival following diagnosis is 10–13 months, with fewer than 5–10% of people surviving longer than five years. Without treatment, survival is typically three months. It is the most common cancer that begins within the brain and the second-most common brain tumor, after meningioma, which is benign in most cases. About 3 in 100,000 people develop the disease per year. The average age at diagnosis is 64, and the disease occurs more commonly in males than females. Tumors of the central nervous system are the 10th leading cause of death worldwide, with up to 90% being brain tumors. Glioblastoma multiforme (GBM) is derived from astrocytes and accounts for 49% of all malignant central nervous system tumors, making it the most common form of central nervous system cancer. Despite countless efforts to develop new therapies for GBM over the years, the median survival rate of GBM patients worldwide is 8 months; radiation and chemotherapy standard-of-care treatment beginning shortly after diagnosis improve the median survival length to around 14 months and a five-year survival rate of 5–10%. The five-year survival rate for individuals with any form of primary malignant brain tumor is 20%. Even when all detectable traces of the tumor are removed through surgery, most patients with GBM experience recurrence of their cancer. A TERT promoter mutation is a genetic event that occurs in the TERT gene and is a significant factor in the development of glioblastoma multiforme (GBM): TERT promoter mutations are found in 60–80% of GBMs. The most common mutations are C228T and C250T These mutations activate telomerase, which is responsible for repairing telomeres and maintaining chromosomal integrity. Cancer cells require telomere lengthening to proliferate infinitely.  TERT promoter mutations are associated with a poor prognosis.  TERT promoter mutations can be detected using liquid biopsies. TERT promoter mutations are also found in other cancers, including oligodendrogliomas, urothelial cancer, melanoma, and thyroid cancers.The prognostic value of TERT promoter mutations is influenced by the status of IDH mutations. TERT promoter mutations and IDH mutations are associated with specific histological glioma subgroups. The combination of TERT promoter mutations and MGMT promoter methylation defines subgroups with noticeable responses to current treatments. GBM is the most common and deadly primary brain tumor in adults, accounting for approximately 45–50% of all primary malignant brain tumors. Hyaluronic Acid Panacur the canine dewormer & Astaxanthin cure Glioblastoma

Leukemia is a group of blood cancers that usually begin in the bone marrow and produce high numbers of abnormal blood cells. These blood cells are not fully developed and are called blasts or leukemia cells. Symptoms may include bleeding and bruising, bone pain, fatigue, fever, and an increased risk of infections. These symptoms occur due to a lack of normal blood cells. Diagnosis is typically made by blood tests or bone marrow biopsy. TET2 mutations are an unfavorable prognostic factor in Acute myeloid leukemia (rapid growth of abnormal cells in the bone marrow and interfere with blood cell production ) patients with intermediate-risk cytogenetics. TET2 mutations are associated with older age, higher white blood cell & blast counts & lower platelet numbers. TET2 mutations are often lost at relapse, but can rarely be acquired. TET2 mutations are common in Chronic myelomonocytic leukemia, occurring in about 45% of cases. Most cases of TET2-mutated Chronic myelomonocytic leukemia have more than one mutant variant. TET2 mutations occur in about 19% of patients with myelodysplastic (undeveloped blood cells) syndromes. TET2 mutations occur in about 12% of patients with myeloproliferative disorders. TET2 mutations occur in about 24% of patients with secondary AML. Secondary acute myeloid leukemia (sAML) is a type of leukemia that develops after a person has had certain blood conditions or cancer treatments TET2 mutations can lead to abnormal epigenetic patterns, which can affect the differentiation and function of hematopoietic stem cells (HPSCs). This can promote the occurrence of leukemia by affecting cell proliferation TET2 deficiency can also lead to an inflamed immune phenotype, which is associated with several inflammation-based diseases, including cardiovascular disease. Leukemia is caused by overfeeding the body with what the bone don't need like fatty foods to prevent leukemia take supplements as Hydrogen Peroxide Vitamin C & Nitric Oxide
https://www.youtube.com/watch?v=3vuoNiV6OY8
What is blood cancer?

Meningitis is a serious infection of the membranes that protect the brain and spinal cord. It can be caused by viruses, bacteria, or fungi. Meningitis is acute or chronic inflammation of the protective membranes covering the brain and spinal cord, collectively called the meninges. The most common symptoms are fever, intense headache, vomiting and neck stiffness and occasionally photophobia. Other symptoms include confusion or altered consciousness, nausea, and an inability to tolerate light or loud noises. Young children often exhibit only nonspecific symptoms, such as irritability, drowsiness, or poor feeding. A non-blanching rash (a rash that does not fade when a glass is rolled over it) may also be present. The inflammation may be caused by infection with viruses, bacteria, fungi or parasites. Non-infectious causes include malignancy (cancer), subarachnoid hemorrhage, chronic inflammatory disease (sarcoidosis) and certain drugs. Meningitis can be life-threatening because of the inflammation's proximity to the brain and spinal cord; therefore, the condition is classified as a medical emergency. A lumbar puncture, in which a needle is inserted into the spinal canal to collect a sample of cerebrospinal fluid (CSF), can diagnose or exclude meningitis. Some forms of meningitis are preventable by immunization with the meningococcal, mumps, pneumococcal, and Hib vaccines. Giving antibiotics to people with significant exposure to certain types of meningitis may also be useful for preventing transmission. The first treatment in acute meningitis consists of promptly giving antibiotics and sometimes antiviral drugs. Corticosteroids can be used to prevent complications from excessive inflammation. Meningitis can lead to serious long-term consequences such as deafness, epilepsy, hydrocephalus, or cognitive deficits, especially if not treated quickly. In 2019, meningitis was diagnosed in about 7.7 million people worldwide, of whom 236,000 died, down from 433,000 deaths in 1990. With appropriate treatment, the risk of death in bacterial meningitis is less than 15%. Outbreaks of bacterial meningitis occur between December and June each year in an area of sub-Saharan Africa known as the meningitis belt. Smaller outbreaks may also occur in other areas of the world. The word meningitis comes from the Greek μῆνιγξ meninx, 'membrane', and the medical suffix -itis, 'inflammation'.

Osteoporosis is a systemic skeletal disorder characterized by low bone mass, micro-architectural deterioration of bone tissue leading to more porous bone, and consequent increase in fracture risk. It is the most common reason for a broken bone among the elderly. Bones that commonly break include the vertebrae in the spine, the bones of the forearm, the wrist, and the hip. Until a broken bone occurs there are typically no symptoms. Bones may weaken to such a degree that a break may occur with minor stress or spontaneously. After the broken bone heals, the person may have chronic pain and a decreased ability to carry out normal activities. Osteoporosis may be due to lower-than-normal maximum bone mass and greater-than-normal bone loss. Bone loss increases after the menopause due to lower levels of estrogen, and after "andropause" due to lower levels of testosterone. Osteoporosis may also occur due to a number of diseases or treatments, including alcoholism, anorexia, hyperthyroidism, kidney disease, and surgical removal of the ovaries. Certain medications increase the rate of bone loss, including some antiseizure medications, chemotherapy, proton pump inhibitors, selective serotonin reuptake inhibitors, and glucocorticosteroids. Smoking and getting an inadequate amount of exercise are also risk factors. Osteoporosis is defined as a bone density of 2.5 standard deviations below that of a young adult. This is typically measured by dual-energy X-ray absorptiometry (DXA or DEXA). Prevention of osteoporosis includes a proper diet during childhood, hormone replacement therapy for menopausal women, and efforts to avoid medications that increase the rate of bone loss. Efforts to prevent broken bones in those with osteoporosis include a good diet, exercise, and fall prevention. Lifestyle changes such as stopping smoking and not drinking alcohol may help. Bisphosphonate medications are useful to decrease future broken bones in those with previous broken bones due to osteoporosis. In those with osteoporosis but no previous broken bones, they are less effective. [needs update] They do not appear to affect the risk of death. Osteoporosis becomes more common with age. About 15% of Caucasians in their 50s and 70% of those over 80 are affected. It is more common in women than men. In the developed world, depending on the method of diagnosis, 2% to 8% of males and 9% to 38% of females are affected. Rates of disease in the developing world are unclear. About 22 million women and 5.5 million men in the European Union had osteoporosis in 2010. In the United States in 2010, about 8 million women and between 1 and 2 million men had osteoporosis. White and Asian people are at greater risk. The word "osteoporosis" is from the Greek terms for "porous bones". The human skeletal system accounts for 14% of total body weight
https://www.youtube.com/watch?v=Cd0YT-OV97c
The Culprit Behind Osteoporosis - Medical Frontiers
https://www.youtube.com/watch?v=JLY7JB1Jmgc
The science of osteoporosis and how common treatments work
https://www.youtube.com/watch?v=eYGkT6OrBk0
Osteoporosis
https://www.youtube.com/watch?v=BsnKZDGSuw0
How Bone Marrow Keeps You Alive

Pancreas Cancer God wants to heal you of Pancreatic cancer which arises when cells in the pancreas, a glandular organ behind the stomach, begin to multiply out of control and form a mass. These cancerous cells have the ability to invade other parts of the body. A number of types of pancreatic cancer are known. The most common, pancreatic adenocarcinoma, accounts for about 90% of cases, and the term "pancreatic cancer" is sometimes used to refer only to that type. These adenocarcinomas start within the part of the pancreas that makes digestive enzymes. Several other type of cancer, which collectively represent the majority of the non-adenocarcinomas, can also arise from these cells. About 1–2% of cases of pancreatic cancer are neuroendocrine tumors, which arise from the hormone-producing cells of the pancreas. These are generally less aggressive than pancreatic adenocarcinoma. Signs and symptoms of the most-common form of pancreatic cancer may include yellow skin, abdominal or back pain, unexplained weight loss, light-colored stools, dark urine, and loss of appetite. Usually, no symptoms are seen in the disease's early stages, and symptoms that are specific enough to suggest pancreatic cancer typically do not develop until the disease has reached an advanced stage. By the time of diagnosis, pancreatic cancer has often spread to other parts of the body. Pancreatic cancer rarely occurs before the age of 40, and more than half of cases of pancreatic adenocarcinoma occur in those over 70. Risk factors for pancreatic cancer include tobacco smoking, obesity, diabetes, and certain rare genetic conditions. About 25% of cases are linked to smoking, and 5–10% are linked to inherited genes. Pancreatic cancer is usually diagnosed by a combination of medical imaging techniques such as ultrasound or computed tomography, blood tests, and examination of tissue samples (biopsy). The disease is divided into stages, from early (stage I) to late (stage IV). Screening the general population has not been found to be effective. The risk of developing pancreatic cancer is lower among non-smokers, and people who maintain a healthy weight and limit their consumption of red or processed meat; the risk is greater for men, smokers, and those with diabetes. There is some evidence that links high levels of red meat consumption to increased risk of pancreatic cancer. Smokers' risk of developing the disease decreases immediately upon quitting, and almost returns to that of the rest of the population after 20 years. Pancreatic cancer can be treated with surgery, radiotherapy, chemotherapy, palliative care, or a combination of these. Treatment options are partly based on the cancer stage. Surgery is one of many treatments that can cure pancreatic adenocarcinoma, and may also be done to improve quality of life without the potential for cure astaxanthin & various dewormers can cure cancer by blocking pdl1 and increasing tp53 gene. Pain management and medications to improve digestion are sometimes needed. Early palliative care is recommended even for those receiving treatment that aims for a cure. Pancreatic cancer is among the most deadly forms of cancer globally, with one of the lowest survival rates. In 2015, pancreatic cancers of all types resulted in 411,600 deaths globally. Pancreatic cancer is the fifth-most-common cause of death from cancer in the United Kingdom & the third most-common in the United States. The disease occurs most often in the developed world, where about 70% of the new cases in 2012 originated. Pancreatic adenocarcinoma typically has a very poor prognosis; after diagnosis, 25% of people survive one year and 12% live for five years. For cancers diagnosed early, the five-year survival rate rises to about 20%. Neuroendocrine cancers have better outcomes; at five years from diagnosis, 65% of those diagnosed are living, though survival considerably varies depending on the type of tumor. Pancreatic cancer is developed by eating too much and overwhelming your pancreas by raising MTOR
https://www.youtube.com/watch?v=52viwfv0cQU
Pancreas Cancer: What Patients Need to Know

https://www.youtube.com/watch?v=ezfwqmKC9Uc
How Gene Editing Is Curing Disease
https://www.youtube.com/watch?v=pihhRgLDbGY
FDA approves groundbreaking sickle cell anemia treatment
https://www.youtube.com/watch?v=0xv0CBujwZU
Teen is one of the first ever to get his genes edited. Why he says the process is 'cool and freaky'
https://www.youtube.com/watch?v=2sAGtqm3o1g
New CRISPR-based sickle cell treatment, explained

Sickle cell disease (SCD), also simply called sickle cell, is a group of hemoglobin-related blood disorders typically inherited. The most common type is known as sickle cell anemia. It results in an abnormality in the oxygen-carrying protein haemoglobin found in red blood cells. This leads to a rigid, sickle-like shape under certain circumstances. Problems in sickle cell disease typically begin around 5 to 6 months of age. A number of health problems may develop, such as attacks of pain (known as a sickle cell crisis) in joints, anemia, swelling in the hands and feet, bacterial infections, dizziness and stroke. Long-term pain may develop as people get older. The average life expectancy in the developed world is 40 to 60 years. It often gets worse within age. All the major organs are affected by sickle cell disease. The liver, heart, kidneys, gallbladder, eyes, bones, and joints also can suffer damage from the abnormal functions of the sickle cells, and their inability to flow through the small blood vessels correctly. Sickle cell disease occurs when a person inherits two abnormal copies of the β-globin gene (HBB) that makes haemoglobin, one from each parent. This gene occurs in chromosome 11. Several subtypes exist, depending on the exact mutation in each haemoglobin gene. An attack can be set off by temperature changes, stress, dehydration, and high altitude. A person with a single abnormal copy does not usually have symptoms and is said to have sickle cell trait. Such people are also referred to as carriers. Diagnosis is by a blood test, and some countries test all babies at birth for the disease. Diagnosis is also possible during pregnancy. The care of people with sickle cell disease may include infection prevention with vaccination and antibiotics, high fluid intake, folic acid supplementation, and pain medication. Other measures may include blood transfusion and the medication hydroxycarbamide (hydroxyurea). In 2023, new gene therapies were approved. A small percentage of people can be cured by a transplant of bone marrow cells. As of 2015, about 4.4 million people have sickle cell disease, while an additional 43 million have sickle cell trait. About 80% of sickle cell disease cases are believed to occur in Sub-Saharan Africa. It also occurs to a lesser degree in parts of India, Southern Europe, West Asia, North Africa and among people of African origin (sub-Saharan) living in other parts of the world. In 2015, it resulted in about 114,800 deaths. The condition was first described in the medical literature by American physician James B. Herrick in 1910. In 1949, its genetic transmission was determined by E. A. Beet and J. V. Neel. In 1954, the protective effect against malaria of sickle cell trait was described.
https://www.youtube.com/watch?v=ezfwqmKC9Uc
How Gene Editing Is Curing Disease
https://www.youtube.com/watch?v=pihhRgLDbGY
FDA approves groundbreaking sickle cell anemia treatment
https://www.youtube.com/watch?v=0xv0CBujwZU
Teen is one of the first ever to get his genes edited. Why he says the process is 'cool and freaky'
https://www.youtube.com/watch?v=2sAGtqm3o1g
New CRISPR-based sickle cell treatment, explained